> Collectively, these results suggest that **age related reductions in the capacity of skeletal muscle to oxidize fat** may partially explain the lower […] fat oxidation in older individuals. Moreover, these studies suggest that aerobic **exercise may be effective in counteracting the age-related reduction** in the oxidative capacity of skeletal muscle. > > Several studies suggest that a reduced capacity to oxidize fat may contribute to fat accumulation […] Aging is associated with reduced fat oxidation […] The age-related reduction in fat oxidation, therefore, may promote the accumulation of total and central body fat. > > Adiposity increases with age. The size of the adipose tissue mass is determined by the balance between the recruitment of lipid substrates (ie free fatty acids) from adipose tissue and their subsequent oxidation by respiring tissues […] The age-related decrease in fat oxidation is related to a reduction in both the quantity and oxidative capacity of respiring tissue. Taken together, these results suggest that an age-related decrease in the capacity of respiring tissues to oxidize fat, rather than decreased free fatty acid release, is a more likely determinant of lipid imbalance and the age-related increase in adiposity. > > Aerobic exercise training increases fat oxidation both at rest and during exercise, possibly by increasing the enzymatic capacity for fat oxidation. Thus, if reduced fat oxidation contributes to age-related changes in adiposity and risk for chronic disease, interventions designed to increase the quantity or oxidative capacity of metabolically active tissue may be effective in improving the health status of elderly individuals. > > [Lipid metabolism in the elderly](https://www.nature.com/articles/1601033.pdf#page=3) MJ Toth and A Tchernof ### [How Muscles Age, and How Exercise Can Slow It](https://www.the-scientist.com/features/how-muscles-age--and-how-exercise-can-slow-it-64708) > To you readers over age 30, we’ve got some bad news for you. Chances are good you’ve already begun losing muscle. And it only gets worse. Up to a quarter of adults over the age of 60 and half of those over 80 have thinner arms and legs than they did in their youth. > > In 1988, Tufts University’s Irwin Rosenberg coined the term “sarcopenia” from Greek roots to describe this age-related lack (penia) of flesh (sarx). Muscle aging likely has several underlying factors, including decreased numbers of muscle stem cells, mitochondrial dysfunction, a decline in protein quality and turnover, and hormonal deregulation. Loss of muscle mass is associated with—and possibly preceded by—muscle weakness, which can make carrying out daily activities, such as climbing stairs or even getting up from a chair, difficult for many seniors. This can lead to inactivity, which itself leads to muscle loss at any age. Thus, older people can enter a vicious cycle that will eventually lead to an increased risk of falls, a loss of independence, and even premature death. > > The good news is that exercise can stave off and even reverse muscle loss and weakness. Recent research has demonstrated that physical activity can promote mitochondrial health, increase protein turnover, and restore levels of signaling molecules involved in muscle function. > #### The role of muscle stem cells > Skeletal muscle consists of multinucleated fibers formed by the fusion of muscle precursor cells, or myoblasts, during embryonic and fetal development and postnatally until the tissue reaches its adult size. Mature fibers are post-mitotic, meaning they do not divide anymore. As a result, in adulthood both muscle growth and repair are made possible only by the presence of muscle stem cells. > > In 1961, Rockefeller University biophysicist Alexander Mauro, using electron microscopy, first described muscle stem cells, calling them “satellite cells” because of their position at the periphery of the muscle fiber.1 Subsequently, researchers have demonstrated that satellite cells are the only cells able to repair muscle—which explains why recovery from muscle injuries among the elderly is slow and often incomplete: the number of satellite cells falls from 8 percent of total muscle nuclei in young adults to just 0.8 percent after about 70 to 75 years of age. > > Of course, a decline of the satellite cells’ ability to divide and repair could also be to blame, but research does not support this idea. In pioneering studies carried out in 1989, biologists Bruce Carlson and John Faulkner at the University of Michigan showed that muscle isolated from a two-year-old rat was repaired faster and better when grafted into two- to three-month-old rats. More recently, we isolated these cells from young and old adults and were surprised to find that elderly human satellite cells grew in culture as well as those from young subjects did. > > The elderly human satellite cells we examined did, however, show dramatic changes in their epigenetic fingerprint, with the repression of many genes by DNA methylation. One gene, called _sprouty 1_, is known to be an important regulator of cell quiescence. Reduced _sprouty 1_ expression can limit satellite cell self-renewal and may partially explain the progressive decline in the number of satellite cells observed in human muscles during aging. Indeed, stimulation of _sprouty 1_ expression prevents age-related loss of satellite cells and counteracts age-related degeneration of neuromuscular junctions in mice. > > https://i.imgur.com/eraU36c.jpg > #### Mitochondrial contributors > Other likely culprits of muscle aging are the mitochondria, the powerhouses of muscle. To work efficiently, skeletal muscle needs a sufficient number of fully functional mitochondria. These organelles represent around 5 percent to 12 percent of the volume of human muscle fibers, depending on activity and muscle specialization (fast-twitch versus slow-twitch). And research suggests that abnormalities in mitochondrial morphology, number, and function are closely related to the loss of muscle mass observed in the elderly. > > In 2013, David Glass of Novartis and colleagues found that markers of mitochondrial metabolism pathways were significantly downregulated as rats aged, and this correlated with the onset of sarcopenia. Although the findings are merely correlative, the timing and near-perfect relationship between decline in mitochondrial gene expression and the onset of sarcopenia provides strong evidence that mitochondrial dysfunction may be driving sarcopenia. The expression of genes and production of proteins that regulate mitochondrial fission and fusion—processes that maintain mitochondrial volume and function—also dropped, suggesting that mitochondrial dynamics are also perturbed during muscle aging. > > As with muscle stem cell decline, the underlying cause of poor mitochondrial health may be gene regulation. In 2016, Alice Pannérec and her colleagues from Nestlé Institute of Health Sciences and Manchester Metropolitan University in the UK examined the transcriptomes of rat and human muscle and found that susceptibility to sarcopenia in both species was closely linked to deregulation of gene networks involved in mitochondrial processes, regulation of the extracellular matrix, and fibrosis, the formation of excess connective tissue in a muscle caused by the accumulation of extracellular matrix proteins. > #### Protein quality control > Even if they eat plenty of protein, older people often cannot maintain muscle mass, probably because their bodies cannot turn proteins into muscle fast enough to keep up with the natural rate of the tissue’s breakdown. Moreover, the muscles of older people undergo lower levels of autophagy, a process that under healthy conditions recycles used and damaged proteins, organelles, and other cell structures. This can result in an imbalance between protein production and degradation that is likely linked to muscle aging. > > There may also be other ways that reduced autophagy may contribute to both muscle loss and muscle weakness during aging. In order to maintain muscle strength, muscle cells must get rid of the intracellular garbage that accumulates over time. In the case of muscle cells, this garbage includes old organelles such as mitochondria and endoplasmic reticuli, clumps of damaged proteins, and free radicals, all of which can become cytotoxic over time. By recycling mitochondria, muscle fibers boost energy production and preserve muscle function. If muscle fibers fail to clear these potentially dangerous entities, they will become smaller and weaker. > #### Blood signals > In 2005, Stanford University stem cell biologist Thomas Rando and colleagues combined the circulation of young and old mice and found that factors in the blood of young mice were able to rejuvenate muscle repair in aged mice. It is now well known that the levels of circulating hormones and growth factors drastically decrease with age and that this has an effect on muscle aging. Indeed, hormone replacement therapy can efficiently reverse muscle aging, in part by activating pathways involved in protein synthesis. > > Moreover, the muscle itself is a secretory endocrine organ. Proteins produced by the muscle when it contracts flow into the blood, either on their own or encased in membrane-bound vesicles that protect them from degradation by circulating enzymes. Bente Pedersen of the Centre of Inflammation and Metabolism and Centre for Physical Activity Research in Denmark was the first to use the term myokine to describe these proteins. Secreted myokines can act locally on muscle cells or other types of cells such as fibroblasts and inflammatory cells to coordinate muscle physiology and repair, or they can have effects in distant organs, such as the brain. > > Although several of these myokines have been identified—in culture, human muscle fibers secrete up to 965 different proteins—researchers have only just begun to understand their role in muscle aging. The first myokine to be identified, interleukin-6 (IL-6), participates in muscle maintenance by decreasing levels of inflammatory cytokines in the muscle environment, while increasing insulin-stimulated glucose uptake and fatty-acid oxidation. Elderly people with high circulating levels of IL-6 are more prone to sarcopenia. Another myokine, insulin-like growth factor 1 (IGF-1), can trigger the swelling of muscle fibers, including after exercise. IGF-1 levels decrease with age, as do levels of the cell-surface receptor that IGF-1 binds to, and mice that overexpress IGF-1 are resistant to age-related sarcopenia. Nathalie Viguerie and colleagues from the Institute of Metabolic and Cardiovascular Diseases at INSERM-Toulouse University in France recently discovered a novel myokine, which they termed apelin.8 The researchers have demonstrated that this peptide can correct many of the pathways that are deregulated in aging muscle. When injected into old mice, apelin boosted the formation of new mitochondria, stimulated protein synthesis, autophagy, and other key metabolic pathways, and enhanced the regenerative capacity of aging muscle by increasing the number and function of satellite cells. As with IGF-1, levels of circulating apelin declined during aging in humans, suggesting that restoring apelin levels to those measured in young adults may ameliorate sarcopenia. > #### Exercise to combat muscle aging > Although the causes of muscle loss are numerous and complex, there is now copious evidence to suggest that exercise may prevent or reverse many of these age-related changes, whereas inactivity will accelerate muscle aging. Earlier this year, for example, Janet Lord of the University of Birmingham and Steven Harridge at King’s College London examined the muscles of 125 male and female amateur cyclists and showed that a lifetime of regular exercise can slow down muscle aging: there were no losses in muscle mass or muscle strength among those who were older and exercised regularly. More surprisingly, the immune system had not aged much either. > > Exercise’s influence on muscle health likely acts through as many mechanisms as those underlying age-related muscle loss and weakness. For example, the number of satellite cells can be increased by exercise, and active elderly people have more of these cells than more-sedentary individuals do. This is the reason why exercise prior to hip and knee surgery can speed up recovery in the elderly. > > Physical activity also affects the muscle’s mitochondria. A lack of exercise decreases the efficiency and number of mitochondria in skeletal muscle, while exercise promotes mitochondrial health. Last year, Caterina Tezze in Sandri’s lab at the University of Padova identified a strong correlation between a decline in the levels of OPA1, a protein involved in shaping the mitochondria, and a decrease in muscle mass and force in elderly subjects, while OPA1 levels were maintained in the muscles of senior athletes who had exercised regularly throughout their lives. > > Exercise can even spur muscle cells to maintain more-youthful levels of gene transcripts and proteins. For example, Sreekumaran Nair from the Mayo Clinic in Rochester, Minnesota, and colleagues found that high-intensity aerobic interval training reversed many age-related differences in muscle composition, including restoring mitochondrial protein levels. And Simon Melov at the Buck Institute for Research on Aging and Mark Tarnopolsky of McMaster University in Canada and their colleagues have found that while healthy older adults (average age 70) had a gene-expression profile that was consistent with mitochondrial dysfunction prior to a resistance exercise training program, in just six months this genetic fingerprint had completely reversed to expression levels comparable to those observed in young subjects. Additionally, exercise improved muscle function: the older adults were 59 percent weaker than the younger adults before training, and only 38 percent weaker afterward. Different types of exercise can trigger variable but specific responses in the muscle. For example, whereas strength training is efficient at making muscle, high intensity interval training in aerobic exercises such as biking and walking had the greatest effect at the cellular level at combating age-related loss and weakness, according to Nair’s work. > > Exercise also appears to influence autophagy. In December 2011, Sandri and his colleagues were the first to report, in mice, that autophagy activity could be boosted by voluntary physical activity, in this case, running on a treadmill. In January 2012, the team of Beth Levine at the University of Texas Southwestern Medical Center confirmed that exercise rapidly increased autophagy activity and that autophagy is required for exercise to have its beneficial effects: physically active mice that were unable to ramp up autophagy did not show any increase in muscle mass, mitochondrial content, or insulin sensitivity after running. > > Finally, exercise can also apparently restore levels of myokines that decline with age. For example, when elderly subjects followed a regular program of physical activity, there was a direct correlation between the improvement in their physical performance and the increase in the level of circulating apelin. Similarly, Ivan Bautmans from Vrije Universiteit Brussel showed that increased circulating levels of inflammation markers correlate with muscle fatigue in geriatric patients, and that resistance training decreased inflammatory myokines in young adults. > > By these mechanisms and others we have yet to discover, exercise can improve overall strength in the elderly, and specifically, the metabolic vigor of skeletal muscle. Being the most abundant tissue in the average human body, accounting for 30 percent to 40 percent of its total mass, muscle is not only critical for locomotion and breathing, but also for glucose, lipid, and amino-acid homeostasis. The age-associated loss of muscle mass and quality thus contributes to the general metabolic dysfunction commonly seen in elderly patients. In older women, one hour of brisk walking produced elevated insulin sensitivity on the following day. Therefore, it is never too late to exercise to try to combat the consequences of muscle aging. ### [How exercise -- interval training in particular -- helps your mitochondria stave off old age](https://www.sciencedaily.com/releases/2017/03/170307155214.htm) > A new study found that exercise -- and in particular high-intensity interval training in aerobic exercises such as biking and walking -- caused cells to make more proteins for their energy-producing mitochondria and their protein-building ribosomes, effectively stopping aging at the cellular level. > They found that while strength training was effective at building muscle mass, high-intensity interval training yielded the biggest benefits at the cellular level. The younger volunteers in the interval training group saw a 49% increase in mitochondrial capacity, and the older volunteers saw an even more dramatic 69% increase. Interval training also improved volunteers' insulin sensitivity, which indicates a lower likelihood of developing diabetes. However, interval training was less effective at improving muscle strength, which typically declines with aging. > The high-intensity biking regimen also rejuvenated the volunteers' ribosomes, which are responsible for producing our cells' protein building blocks. The researchers also found a robust increase in mitochondrial protein synthesis. Increase in protein content explains enhanced mitochondrial function and muscle hypertrophy. Exercise's ability to transform these key organelles could explain why exercise benefits our health in so many different ways. > > Muscle is somewhat unique because muscle cells divide only rarely. Like brain and heart cells, muscle cells wear out and aren't easily replaced. Functions in all three of those tissues are known to decline with age. "Unlike liver, muscle is not readily regrown. The cells can accumulate a lot of damage," Nair explains. However, if exercise restores or prevents deterioration of mitochondria and ribosomes in muscle cells, there's a good chance it does so in other tissues, too. ### [Cellular stress 'resets lifespan profoundly'](https://www.medicalnewstoday.com/articles/320009.php) > Surprising results from a recent study show that stressing a cell can reverse signs of cellular aging. The findings might open doors to more successful ways to slow the aging process. > The researchers behind the new study, which was headed up by senior study author Prof. Richard I. Morimoto, put the cells' mitochondria — or the so-called powerhouses of the cell — under mild duress. > > What they found came as a surprise: under these conditions, mitochondria sent out signals to the protein machinery, preventing it from failing. This, in turn, reduced the buildup of badly packed proteins. ### [Slow walking speed in midlife linked with faster aging](https://www.medicalnewstoday.com/articles/326648.php) > New research finds that people who tend to walk more slowly at the age of 45 present with signs of premature accelerated aging, both physically and cognitively. > > https://i.imgur.com/CsnrdWd.png > > New research suggests that a slower walking speed in midlife may be a marker of accelerated aging. Walking speed may be a powerful predictor of lifespan and health. ### [Dietary restriction through intermittent fasting](https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6231542/#S7title) > An additional DR regime, called intermittent fasting (IF), requires cycles of ad libitum food availability and complete food deprivation. Like the previously described regimes, IF also extends lifespan in diverse organisms such as worms and mice. Intriguingly, mice on IF can consume the same or even more calories than ad libitum fed mice and still reap the beneficial effects of DR. This observation suggests that IF induces some form of “starvation signals” that are more important than the amount of calories eaten. ### [Targeting Mitochondria to Counteract Aging: Caloric restriction](https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5867886/#sec4-genes-09-00165title) > Caloric restriction: From C. elegans [112] to primates [113], caloric restriction (CR) has been shown to counteract age-related decline and to increase lifespan [114,115]. The occurrence of metabolic, neurological, and cardiovascular diseases was decreased by a 20–40% reduction in caloric intake [116]. While reducing the overall metabolism, CR was reported to shift the metabolism from carbohydrate to fatty acid metabolism, strongly stimulating mitochondrial energy production [75]. CR also enhanced the activity of complex IV as well as ROS-producing complexes I and III in the brains of mice, causing elevated MnSOD activity and redox capacity in CR brains [117]. Consistent with that, low levels of mitochondrial ROS were reported to initiate health-promoting antioxidant defense mechanisms and increase life span via so-called “mitohormesis” [118]. The effect of CR is not just limited to mitochondrial respiration. In liver mitochondria of mice, Ca2+ retention capacity was enhanced strongly by CR in comparison to ad libitum diet, offering protection from ischemia/reperfusion damage, a condition frequently observed in age [119]. Additionally, CR decreased the activity of cyclophilin D through deacetylation, resulting in the increased capacity of mitochondria to buffer Ca2+ and enhanced tolerance against excitotoxicity in rat brain mitochondria [117]. ### [Evidence for a carnivore diet](https://www.kevinstock.io/health/evidence-for-a-meat-based-diet/) > #### Evidence for a Meat-Based Diet: 1.0 – Evolution > Throughout human evolution, selective pressures fueled a persistent feedback loop between climate, food availability, and the advantages of a large brain. > > Archaeological and palaeontological evidence indicate that increased meat consumption underpinned our stunning divergence from our primate ancestors. > > Animal fat, with its ready-made source of long-chain fatty acids, powered the human brain to expand to 4X the size from that of our hominid ancestors. > >From Homo habilis to erectus to sapiens, meat gradually became the staple food of humans. > > #### Fossil Record > Meat was the fork that diverged humans on our path to the top of the food chain. > #### A Hunter’s Body > Not only are our internal organs and tissues evidence for a meat-based diet, but so is the adaptations of our outer body. > > Humans are the only living primate adapted for endurance running. > > Everything from our balancing vestibular system and nuchal ligament, to our toe alignment and shock-absorbing foot structure, to our scarce body hair and cooling eccrine sweat glands, to our long lower limbs evidence a persistence hunter physique. Bodies optimized for running and hunting down prey with unparalleled stamina. I have tried to implement the ketogenic diet several times over the past years: https://www.health.harvard.edu/blog/ketogenic-diet-is-the-ultimate-low-carb-diet-good-for-you-2017072712089 What was happening to me was that I needed so much fat to be satiated, that it was accumulating in my belly. Presumably I have some dysfunction with fatty liver, lipid fat turnover rate, metabolism, and possibly altered gut microbiota (perhaps from antibiotics) as an underlying cause or contributing factor. Also I think my age and the swelting tropical climate are other factors limiting my ability to exercise sufficiently to correct the metabolic imbalances in my body. Note that the article you cited mentions that we are natural runners. We need to sprint a lot and jog. I used to do that a couple of hours a day, but I had lost that and become sedentary in the Philippines. I am trying to do more consistent, daily, sprinting and jogging now here, but I do get tired of the heat and I get low energy especially if I had insufficient intake of calories. Sometimes I just can’t feel right eating only meat and need more carbs. This is why I want to move asap to a cooler, drier, sunny climate (without intense Winters) so I can maximize my outdoor athletics. Note I have found the (wild caught, not farm raised) Norwegian Rainbow Trout is one of the best meats I can eat to have a great balance between being satiated and not accumulating belly fat. The Omega 3s appear to be quite abundant in that fish. I found that eating the leg and thigh of chicken (which are very fatty) would accumulate belly fat. But this was much improved when I ate the free range, wild chicken, which have much less fat in their legs and thighs. I remember reading that cows and chickens which are not eating in the wild (e.g. grass fed) having nearly no Omega 3 and all Omega 6. Whereas, wild animals have as much Omega 3 as 6. So apparently it is not sufficient just to eat meat and reduce carbs. We must also increase sprinting and jogging, eating only wild animals that forage in the wild, and when we do eat carbs those carbs should be both minimized and include some unprocessed carbs such as an unripe banana, blueberries, and maybe whole rolled oats from oatmeal or at least sourdour whole gain bread for the probiotic effects. Unripe because fructose is immediately converted to belly fat. Note I have been eating frozen, unsweetened blueberries with unsweetened yogurt the past few nights and the blueberries are an amazing antioxidant. I feel some effects on my gut. So I am curious to see how this is going to play out over time. Also I do get low energy, forehead-on-the-keyboard sometimes in the middle of the day (presumably from my gut health issue because it usually coincides with recovery days from gym and I get this insatiable hunger that can’t be satiated unless I eat so much that my belly fat is engorged). If I try to exercise through these episodes it is very arduous, low-energy, etc.. The iodine has been helping me battle this. It is not a perfect solution, but it does help a lot. Before all I could do was wallow in bed for 2 days after every gym workout. Note as I sustain my gym workouts and sprinting, and stay outdoors a lot, I seem to stabilize and the episodes become less frequent or pronounced. In short, I hope I am on the verge of being able to get my health corrected to a sustainable regimen. I am still working on all the variables, and including wanting to be back in the USA asap. This several years health battle has been an enormous pita and loss of a huge chunk of my life and productivity. I learned a life lesson about who and want to expose my body to. Note we still need green veggies in some amounts to help our bodies digest meat: https://examine.com/nutrition/scientists-just-found-that-red-meat-causes-cancer--or-did-they/ https://chriskresser.com/red-meat-cancer-again-will-it-ever-stop/ ### [The Impact of Dietary Iodine Intake on Lipid Metabolism in Mice](https://www.researchgate.net/publication/45283610_The_Impact_of_Dietary_Iodine_Intake_on_Lipid_Metabolism_in_Mice) > The present study has been designed to investigate the impact of dietary iodine intake on lipid metabolism in mice, including iodine deficiency and iodine excess […] In conclusion, dietary iodine intake may affect the metabolism of serum lipids. Hypothyroid function induced by iodine deficiency may be responsible for the changes of lipids. Higher iodine intake might benefit lipid metabolism. ### [Effects of the Long-Term (7-9 Months) Feeding of Iodine-Enriched Eggs on Lipid Metabolism of Rats](https://www.jstage.jst.go.jp/article/jnsv1973/29/1/29_1_23/_article) > The effect of long-term (7-9 months) feeding of a diet containing iodine-enriched eggs, which had considerably higher amounts of iodine than ordinary eggs, was studied on the lipid metabolism in rats […] The long-term feeding with iodine-enriched eggs did not influence body weight gain, food intake, voluntary running activity, weights of several organs and adipose tissues, and lipid concentrations in several organs and tissues. However, the serum lipid levels were decreased after the feeding of iodine-enriched eggs. A decrease in triacylglycerol (TG) level was significant (p<0.05). The long-term feeding with iodine-enriched eggs resulted in a reduced hepatic-intestinal TG production rate and also an enhanced TG removal rate from blood. Furthermore, skeletal muscle and adipose tissue lipoprotein lipase activity was significantly elevated with the feeding of iodine-enriched eggs. These results suggest that the lowering effect of blood TG level with feeding of iodine-enriched eggs may be mediated by either a reduced hepaticintestinal TG production or an enhanced peripheral removal of circulating TG. Serum total iodine concentration was 5 times higher in the iodine-enriched egg fed rats as compared to controls, but the serum concentrations of thyroid stimulating hormone and thyroid hormones as well as the thyroid total iodine content were not different between the two dietary groups of animals. ### [Why I’m Taking Iodine](https://revealingfraud.com/2019/06/health/why-im-taking-iodine/) > Iodine (Lugol’s 2% iodine at amazon.com) and Selenium are probably the most powerful and yet most neglected and forgotten mineral supplements. Ironically, iodine boosts memory and IQ! Iodine kills fungus, it’s also a broad spectrum antibiotic, and so it fell out of favor in the 1950’s with the rise of more expensive and more profitable fungal based antibiotics. Iodine and Boron both detox fluorides; and iodine fixes and cures 180 conditions (listed below) associated with both fluoride toxicity and iodine deficiency. Iodine is an oxidant, (selenium the protective antioxident), and nourishes every cell in the body, especially the thyroid gland. Iodine cures goiter. Iodine concentrates in the sex organs of both sexes, boosting testosterone, regulating female hormones, and cures breast cancer. Iodine also cured diabetes in half the people on the “high iodine protocol”, which I will go over. > > This report on iodine reveals the fraud of the medical establishment’s recommendation of less than 1mg iodine/day, the fraud of MRSA, the fraud of fungal based and fluoride based antbiotics, and the fraud of water fluoridation. ### [How I Increased my Brainpower, Memory, & Focus](https://revealingfraud.com/2019/08/health/how-i-increased-my-brainpower-memory-focus-4-6-16/) > They are saying iodine at 150 mcg can boost IQ by 15 points. I’m taking 50 mg/day, or 50,000 mcg of iodine. > > The day I took iodine was a game changer in my head. Suicidal thoughts went away. My memory increased. My focus increased. I could study for long periods. I could concentrate better. I could remember what I was reading much better. I was much less angry. I could think about the IRS harassment, and prior business thefts, and not spin out in anger. This? From a mineral? Wow. What the hay? > > Iodine removes fluoride (which makes people dumber, and here’s a study on that from Harvard!): > > > “The average loss in IQ was reported as a standardized weighted mean difference of 0.45, which would be approximately equivalent to seven IQ points for commonly used IQ scores with a standard deviation of 15.* Some studies suggested that even slightly increased fluoride exposure could be toxic to the brain. Thus, children in high-fluoride areas had significantly lower IQ scores than those who lived in low-fluoride areas. “ > > http://www.hsph.harvard.edu/news/features/fluoride-childrens-health-grandjean-choi/ > > Iodine also removes bromine, aluminum and mercury. Iodine at 3mg/day may cure breast cancer?! Iodine cures goiter, and boosts the thyroid, but is needed in all body tissues. > > Iodine was another mineral, like boron, that you can take a lot of, like 500 to a thousand times more than the minimum RDA, and appears very beneficial. ### [The Vitamin or Mineral Deficiency Causing Your Condition](https://revealingfraud.com/2019/08/health/the-vitamin-or-mineral-deficiency-causing-your-condition/#comment-577) > So, my first thought, is all the minerals that help the nerves, and there are many. > Magnesium is relaxing to the nerves. > Zinc is relaxing to the nerves. > Copper helps rebuild the myelin sheath, the fatty layer around the nerve, as does iodine, and B12, and DMSO/MSM sulfur. > > Then, there is the thought of general detoxing of those heavy metals that are particularly bad for the nerves, as follows: > lead, which is detoxed by zinc > fluoride, which is detoxed by iodine, and boron > mercury, which is detoxed by iodine, and selenium > aluminum, which is detoxed by silica. > > Also note, there are three other mainstream macro minerals that the nerves rely on: calcium, salt (sea salt) and iron. Note, iron is best metabolized with copper and B12, both of which were already listed. > > For sleep, which is listed as part of the disorder, zinc at night helps with. So that’s zinc listed for 3 reasons now. Generally, people who eat vegan, or not enough meat, get low in zinc. ### [Why I take all the minerals](https://revealingfraud.com/2019/04/health/why-i-take-all-the-minerals/) > I’m taking the minerals for quite a few reasons: > > 1. The soils our food is grown in are notoriously deficient in minerals. > 2. Sugar has no minerals, and uses up the little minerals we have. > 3. I’ve noticed more benefits from minerals than vitamins. > 4. Taking a few minerals can lead to depletion in the other more trace minerals, so it’s better to take them all. > 5. Many trace mineral supplements don’t have enough. > 6. Even a multi mineral supplement does not have nearly enough. > 7. I have discovered that you have to take the minerals at different times to avoid nausea, especially taking zinc by itself at night. > > Iodine is my most important mineral supplement. Iodine fixes 180 conditions that are linked to iodine deficiency and fluoride toxicity, and iodine detoxes fluoride. Fluoride also induces iodine deficiency. They block each other. Iodine makes you smarter and fixes about 14 brain conditions, including depression. Iodine is an anti fungal, and anti bacterial. Iodine boosts testosterone. ### [Can herpes contribute to cognitive decline?](https://www.medicalnewstoday.com/articles/306175.php) > Worldwide, an estimated 417 million people under the age of 50 have the type of herpes simplex virus typically associated with genital herpes. Given that this is a significant chunk of the world's population, it would be disquieting if the virus was linked to something more nefarious. Prepare to be disquieted: a new study finds that certain chronic viral infections - including the herpes simplex virus - may contribute to cognitive decline in healthy older adults. ### [Google’s Digital Book Burn: Alternative Medicine Content Now Vanishingly Rare, Despite 1 Billion Health Searches a Day](https://www.sgtreport.com/2019/08/googles-digital-book-burn-alternative-medicine-content-now-vanishingly-rare-despite-1-billion-health-searches-a-day/) > With about 1 billion health searches happening on Google every day, it’s not hard to understand why censoring natural health information serves a multitude of interests, not the least of which is Big Pharma’s bottom line. > > A new analysis released by Bob Troia, Twitter handle Quantified Bob, reveals how health sites have been decimated by Google in the period tracked from April 15th, 2018 to August 15th, 2019. > > He posted the following graphic on Twitter on August 25th, 2019, showing as high as a 99.87% decrease in visibility through Google search results, and a 81.14% drop for Greenmedinfo.com. ### [What are straight-up facts people won’t swallow?](https://revealingfraud.com/2019/08/health/what-are-straight-up-facts-people-wont-swallow/) > 1. Most people really don’t care about you hardly at all. They have their own problems. > 2. The person with an IQ (intelligence quotient) of 100 is average, and quite stupid, and half of all people are dumber than that. > 3. Evolution is a lie. > 4. Politicians are far more corrupt than you think. > 5. The media constantly lies. > 6. Doctors accidentally kill about 1,000,000 people each year. > 7. Mass shootings only kill about 100 people each year. > 8. [redacted] > 9. Vaccines contain many deadly poisons and probably kill about 30,000 each year, and dumb down the entire population. > 10. There are plenty of conspiracies that have been proven true over the years. Conspiracy is just two or more people plotting crimes. > 11. All gun control laws violate the second amendment […] > 12. Fluoride is a deadly nerve toxin that lowers IQ by about 7 points, from the water alone. Teflon and goretex are primarily fluoride. Putting fluoride in the water should be considered the crime of mass poisoning and a terrorist plot. Iodine deficiency lowers IQ by about 15 points. > 13. Getting on the high iodine protocol as a mother who then gets pregnant increases the baby’s IQ by about 40 points. > 14. The government and medical establishments are both poisoning you, and both don’t want you to be smarter, because then you are harder to control and lead and steal from. > 15. Healthy people put doctors out of business. > 16. [redacted] > 17. You can cure just about every single disease simply through taking enough vitamins and minerals, as long as the rest of your diet does not consist of vodka and doughnuts. > 18. Democrats were the party of slavery in the Civil War. > 19. Democrats are still the party of slavery; they just call it socialism and higher taxes on the rich. > 20. The Nazis were socialist democrats, IE, the “National Socialist German Workers’ *Party”. * > 21. Democrats and black people are the most racist groups of people in the world. > 22. Blacks have the lowest IQ’s on Army tests. The next highest group are the Arabs and Indians, then white people are in the middle, then the Asians, and then the Jews at the top. This is so politically incorrect, it’s nearly impossible to say anywhere. > 23. Democrats probably suffer from brain damage from all the poisoning being done to people; they are notoriously hypocritical and severely lacking in self awareness, and they don’t remotely practice what they preach. > 24. 70% of people in America take prescription medicines that reduce their brainpower and make them dumber. > 25. [redacted] and the world is run by the world bankers; forget all the other “illuminati” / CFR groups, they are minor clubs compared to the world bankers. > 26. None of the world bankers appear on the Forbe’s richest lists. > 27. Usury is any interest rate, not an “excessive interest rate”. > 28. The borrower is the servant to the lender. > 29. The USA was overthrown at the time of the Constitution, because the Articles of CON-Federation were overthrown and replaced by the Constitution to pay down debts to the world bankers. A government that serves the lender cannot serve the people. > 30. [redacted] > 31. [redacted] > 32. Censorship exists everywhere, [even on quora.com](https://steemit.com/politics/@anonymint/end-of-democracy-and-a-civil-cold-war-approaches) > 33. If the media censors it, people won’t believe it.
Originally posted here: https://steemit.com/health/@anonymint/intense-exercise-and-intermittent-fasting-combats-deleterious-health-as-we-age
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